Inhibitory kinesiotaping has no effect on post-stroke spasticity: Prospective, randomised, controlled study

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dc.authoridkaracan, ilhan/0000-0002-7462-1358
dc.authoridTURKER, KEMAL SITKI/0000-0001-9962-075X
dc.contributor.authorZincirci, Dilara Ekici
dc.contributor.authorYurttutmus, Zeynep
dc.contributor.authorTürker, Kemal Sıtkı
dc.contributor.authorKaracan, İlhan
dc.date.accessioned2024-09-11T19:51:03Z
dc.date.available2024-09-11T19:51:03Z
dc.date.issued2024
dc.departmentİstanbul Gelişim Üniversitesien_US
dc.description.abstractObjective: Motor neuron pool activity is high in spasticity. The effect of inhibitory kinesiotaping (KT) on spasticity is unclear. The aim of this study is to investigate the effect of inhibitory KT on spasticity after stroke. Methods: Fifty stroke patients with ankle plantarflexor spasticity were randomised to intervention (27) and control (23) groups. Inhibitory KT was applied to the triceps surae muscle in the intervention group and sham KT to the Achilles tendon in the control group. Inhibitory and sham KT were applied for 72 h with a combined conventional rehabilitation programme. Spasticity was assessed at baseline and 72 h after KT using three instruments: Modified Ashworth Scale (MAS), Homosynaptic Post -Activation Depression (HPAD) reflecting the level of motor neuron pool activity, and joint torque as a measure of resistance to passive ankle dorsiflexion. Results: The baseline MAS score, HPAD levels and dorsiflexion torque of the two groups were not significantly different. The change in MAS score was -3.7 +/- 17.5 (p = 0.180) in the intervention group and 3.6 +/- 33.3 (p = 0.655) in the control group. The change in dorsiflexion torque was -0.3 +/- 16.1 kg m (p = 0.539) in the intervention group and 8.0 +/- 24.1 kg m (p = 0.167) in the control group. The change in mean HPAD was 8.7 +/- 34.7 (p = 0.911) in the intervention group and 10.1 +/- 41.6 (p = 0.609) in the control group. Conclusions: The present study showed that inhibitory KT has no antispastic effect in stroke patients.en_US
dc.identifier.doi10.1016/j.jbmt.2024.01.029
dc.identifier.endpage196en_US
dc.identifier.issn1360-8592
dc.identifier.issn1532-9283
dc.identifier.pmid38763562en_US
dc.identifier.scopus2-s2.0-85184045098en_US
dc.identifier.startpage191en_US
dc.identifier.urihttps://doi.org/10.1016/j.jbmt.2024.01.029
dc.identifier.urihttps://hdl.handle.net/11363/7728
dc.identifier.volume38en_US
dc.identifier.wosWOS:001186350200001en_US
dc.identifier.wosqualityN/Aen_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.language.isoenen_US
dc.publisherElsevieren_US
dc.relation.ispartofJournal of Bodywork And Movement Therapiesen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.snmz20240903_Gen_US
dc.subjectH -reflexen_US
dc.subjectHemiplegiaen_US
dc.subjectMuscle toneen_US
dc.titleInhibitory kinesiotaping has no effect on post-stroke spasticity: Prospective, randomised, controlled studyen_US
dc.typeArticleen_US

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